Adult Paediatric On-Call Training

Foundation 15 min read

Adult Paediatric On-Call Training

By Emily, Sammy Reed · 2024

Specialist Topics

Authors: Emily and Sammy (adapted from L. Murphy, S. Reed, and M. Revell) Source: Adult Paeds On Call Training.pptx (56 slides) Learning Level: Intermediate to Advanced


Learning Objectives


Part 1: Scope of On-Call Practice

1.1 Appropriate Call-Outs (Indications for On-Call Physio)

The following are legitimate reasons for on-call physiotherapy attendance:

1.2 Inappropriate Call-Outs

The following are not appropriate indications for on-call physiotherapy:

1.3 What to Ask on the Phone

When receiving an on-call referral, gather the following information:

1.4 Contraindications and Precautions to Respiratory Physiotherapy

Contraindications / PrecautionsNotes
Undrained pneumothoraxAbsolute contraindication
Pulmonary embolism (PE)
Clotting abnormalitiesCheck platelets and INR
Metabolic bone disease
Osteopenia / osteoporosis +/- previous fractures
FatigueEspecially in neuromuscular patients
Basal skull fractureNo NP suction or NP airway

Part 2: Respiratory Assessment Framework

2.1 Systematic Assessment

Use an A-E (Airway, Breathing, Circulation, Disability, Exposure) systematic approach for respiratory assessment.

2.2 Normal Paediatric Values

Respiratory Rate

AgeBreaths per Minute
Neonate (0)40-60
1-3 years20-30
3-6 years20-30
> 6 years15-20
Adult15

Heart Rate

AgeAverageMin / Max
Neonate (0)140100-200
6 months13090-170
1 year12080-150
7 years10070-135
14 years8555-120

2.3 PEWS Score (Paediatric Early Warning Score)

Early warning scores are generated by combining the scores from a selection of routine observations. Different observations are selected for children and adults due to their naturally different physiological responses. If a child’s clinical condition is deteriorating the score will usually increase, giving an early indication that intervention may be required. Early intervention can fix problems and can avoid the need to transfer a child to a higher level of care, thus avoiding or reducing harm.

Updated (2026): It is important to note that the only large RCT of a Paediatric Early Warning System — the EPOCH trial (Parshuram CS et al., 2018) — evaluated implementation of BedsidePEWS across 21 hospitals in 7 countries (144,539 patient discharges) and found that PEWS implementation did NOT decrease all-cause mortality. PEWS remains widely used as a structured framework for recognising deterioration and triggering escalation, and pre-/post-implementation studies have shown associations with reduced mortality and unplanned code events (Trubey et al. 2022, low-quality evidence). However, learners should be aware that the strongest available trial-level evidence does not demonstrate a mortality benefit. References: Parshuram CS et al. JAMA. 2018;319(10):1002-1012. DOI: 10.1001/jama.2018.0948. Trubey R et al. Resuscitation Plus. 2022;11:100262.

2.4 CXR Interpretation

Key paediatric CXR features:

2.5 Blood Results — Key Values

TestNormal Range / Threshold
Platelets> 50 (> 70 for procedures)
INR1.1
Potassium3.5 - 5
Haemoglobin (Hb)> 70 (> 110-140 normal)
CRP< 1
WCC5 - 13
Alkaline Phosphatase< 500

2.6 Blood Gas Interpretation

Types of Blood Gas

Key principles:

  • Find out what is normal for the patient
  • Trends are important
  • Know the type of gas
  • Squeezed samples may have more venous blood

Acid-Base Interpretation

Normal Blood Gas Values

ParameterABG (< 2 yr)ABG (> 2 yr)CBGVBG
pH7.3 - 7.47.34 - 7.457.28 - 7.387.25 - 7.3
pCO2 (kPa)4.47 - 4.574.7 - 6.04.5 - 5.05.0 - 6.7
pO2 (kPa)8 - 1310 - 135 - 73.5 - 5.5
HCO3 (mmol/L)17 - 2418 - 2716 - 22
BE-2 to +2-2 to +2-2 to -4

Blood Gas Quiz (Worked Examples)

CaseTypepHCO2O2HCO3BEInterpretation
1ART7.20710.910.7262Respiratory acidosis with nil compensation
2CBG7.5137.3574418Metabolic alkalosis with partial compensation
3CBG7.262.2511.910.7-18.4Metabolic acidosis with partial compensation
4ART7.2110.3730.2-0.8Respiratory acidosis with partial compensation
5CBG7.3510.112.2318.8Mixed: either metabolic alkalosis or respiratory acidosis, fully compensated

Part 3: Paediatric Anatomy and Physiology

3.1 Key Differences from Adults

  1. Nose breathing: Babies are obligatory nose breathers until age 4-6 months. If their nasal passage is at all occluded, their work of breathing is increased.

  2. Airway compliance: Adult airways are more cartilaginous than children’s. This makes children’s airways more floppy/malacic and therefore more at risk of collapse, obstruction, and secretion retention.

  3. Rib mechanics: There is no bucket-handle action and external intercostal muscles are poorly developed. Inspiration is less efficient as rib movement is reduced in both the AP and transverse planes.

  4. Cardiac space: The younger the child, the more space their heart takes up in the thoracic cavity.

  5. Diaphragm shape: Due to horizontal insertion, children’s diaphragms are less dome-shaped, meaning they cannot flatten as much as adults to increase tidal volumes. To increase minute volume, their RR must be higher.

  6. Heart rate and muscle fibres: Adult HRs are lower than children’s due to lower oxygen demands. Children have fewer fast-twitch muscle fibres, meaning they are more prone to fatigue and bradycardias.

  7. Respiratory reserve: Children have fewer fatigue-resistant muscle fibres, which means they have reduced respiratory reserve. A significant sign of fatigue is apnoeas, so they are more likely to have a respiratory arrest than adults.

3.2 Signs of Respiratory Distress

Observable signs:

Appearance:

Sounds:

Behaviour:

Objective markers:


Part 4: Common Respiratory Conditions

4.1 Bronchiolitis

Key features:

Management (per NICE Guidelines):

No physiotherapy unless significant co-morbidities or intubated

Updated (2026): Palivizumab is more accurately described as passive immunisation with a monoclonal antibody rather than a “vaccination”. From 2025, nirsevimab (a longer-acting, single-dose monoclonal antibody) has begun replacing palivizumab in NHS England. See JCVI statement, 11 September 2023.

4.2 Lower Respiratory Tract Infection (LRTI)

4.3 Neuromuscular Conditions

Examples: SMA, Duchenne’s Muscular Dystrophy, Myasthenia Gravis

Key features:

4.4 Neurodisability

Definition: Impairment involving the nervous system (umbrella term)

Examples: Cerebral palsy, autism

Key features:

4.5 Other Conditions Encountered

The following conditions may be encountered on call (familiarity expected):

CF, PCD, Bronchiectasis, CLD/BPD, Sickle Cell, Asthma, T21, SMA, Di-George Syndrome, Leukodystrophy, Apert Syndrome, Fanconi’s Anaemia, Congenital Myopathy, General Developmental Delay, Ataxia Telangiectasia, Idiopathic Scoliosis, HIE, Batten’s Disease, Dravet’s Disease, Krabbe’s Disease, Woree Syndrome, Q22


Part 5: Medications and Nebulisers

5.1 Bronchodilators

TypeExamplesMechanism
Beta-2 adrenoreceptor agonistsSalbutamolActivates the beta-2 receptors on the muscles surrounding airways, causing relaxation of smooth muscle
Non-beta-2 agonistsAdrenalineStimulates alpha and beta receptors in sympathetic nervous system, inhibits an enzyme in smooth muscle. Note: also increases HR and BP by increasing cardiac output
AntimuscarinicsAtrovent (ipratropium)Blocks muscarinic receptors found on smooth muscle, causing dilation

Physiotherapy relevance:

5.2 Hypertonic Saline (3% and 7%)

5.3 Mucolytics

MucolyticMechanismTiming Before Physio
N-Acetylcysteine (NAC)Breaks the disulphide bonds in mucoproteins holding sputum together; reduces viscosity; decreases sputum hypersecretion30 minutes pre-physio
DNaseContains deoxyribonuclease (enzyme); breaks down long DNA molecules in sputum into smaller fragments; reduces sputum viscosity45 minutes to 1 hour pre-physio

5.4 Nebulised Antibiotics


Part 6: Treatment Options

6.1 Available Treatment Modalities

6.2 Suction

TypeNotes
Oropharyngeal (OP)Appropriate on call
Nasopharyngeal (NP)Appropriate on call; check for contraindications (basal skull fracture)
YankauerFor oral suction

Ensure correct size and depth of catheter. NOT deep suction (this is a separate competency).

6.3 Positioning

Considerations include:

Important considerations for positioning:

  • Subluxed / dislocated hips
  • Scoliosis
  • Fractures
  • Osteopenia
  • Pressure sores
  • Pain

6.4 Drying Agents (Glycopyrronium vs Hyoscine)

When to consider:

AgentRouteKey Properties
Glycopyrronium (Glyco)Oral / IVEasy to titrate (short half-life). Time around feeds: 1 hour pre-feed or 2 hours post-feed for effectiveness
HyoscinePatchDo NOT cut the patch. Replace every 72 hours. Slow to titrate up and down

6.5 MDT Support

Remember to consider what the wider MDT can aid with (e.g. medical team for escalation, nursing for suctioning regimes, medical team for ventilatory support changes).


Part 7: Inhaled Nitric Oxide (iNO) and Physiotherapy

7.1 iNO Machine — Key Readings

Top of the machine:

DisplayMeaning
Top left numberFiO2 (may differ from what the vent or HFOV is reading due to oxygen blending with nitric). The FiO2 on the nitric machine is the accurate reading, not the number on the vent/HFOV
Middle numberIgnore
Top right numberAmount of nitric oxide in ppm (parts per million). Starts at 20 and is slowly weaned down over a few days

Bottom of the machine:

ComponentInstructions
Bag connectionConnect the bag to the iNO machine instead of the oxygen flow meter at the bedspace
Middle dialTurn to whatever the nitric is reading (top right number in ppm). Complete this as you are about to disconnect — the gas is expensive so you do not want it escaping. When you reconnect the patient to the machine, turn the dial back to 0
Right hand side (flow meter)Turn up to 10 for small bags and 15 for large bags

7.2 iNO Safety

CRITICAL: Do NOT disconnect iNO to perform MHI.

Disconnecting can cause:

  • A sudden rise in pulmonary artery pressure
  • Severe strain on the right-hand side of the heart
  • Potential hypoxaemia

Instead, connect the bag to the iNO machine.

7.3 Physiotherapy Benefit with iNO


Part 8: Clinical Scenarios

8.1 Case Study 1: SMA Type 1 — LRTI with Secretion Retention

Patient: 21-month-old male

Presentation: Admitted to the ward with parents from ED. 24-48 hour history of increased lethargy, thick yellow secretions at home.

Background: Spinal muscular atrophy (SMA) type 1, recurrent hospital admissions for LRTIs, growing pseudomonas.

What to Ask the Parents

Home regimen (when well — mum reports once daily in the morning):

  • 3% hypertonic saline nebuliser
  • Cough assist (2 x 2 ASL)
  • Suction: size 7, to 16 cm depth
  • All completed by mother

Assessment Findings

Airway Clearance Techniques

Key Considerations for Neuromuscular/Neurodisability Patients

IMPORTANT: Children with neuromuscular/neurodisability disorders cannot compensate for respiratory failure in the same way as other children.

They may appear: withdrawn, pale, and sweaty

They may NOT show:

  • Increased respiratory rate
  • Increased abdominal work or accessory muscle use

Treatment Session Plan

8.2 Case Study 2: Cerebral Palsy with Viral Illness

Patient: 8-year-old male

Presentation: Admitted to ward with a 3-day history of increased thicker secretions and a temperature. Parents and sibling also unwell at home. Admitted overnight, now with increasing Optiflow requirement and requiring frequent nasal suction. Large volumes of saliva, audible secretions; nursing staff struggling to clear secretions.

Background: Cerebral palsy GMFCS 5, CLD, scoliosis, PEG fed, dystonia, known to various community services. Extensive drug history.

Investigations: Flu A positive.

Further history from mother:

  • Unwell for 3 days
  • Vomiting and high fevers (highest 38 degrees)
  • Mother also became unwell with fever and headache
  • Got worse and mother called ambulance
  • Had salbutamol in ambulance and ED
  • Reviewed in 2 hours post burst

What to Gather

Management Considerations


Escalation Pathways

When to escalate to the medical team:

Always ensure the NIC/medical team is aware of the patient’s status and your assessment findings.